Intermittent fasting practiced by celebrities such as Elon Musk, Jennifer Aniston and Kourtney Kardashian does not make you younger, according to a new study.
Scientists said there is no concrete evidence to suggest missing out on food for long periods is effective at preventing aging.
According to researchers, two treatments, one of which targets how cells produce energy and another that targets a growth hormone, are also effective at preventing aging.
Reality star Kourtney Kardashian said she would not eat past 7 pm at night and then waited until around 10:30 am or 11 am the next day.
Now scientists are saying this approach does not slow the aging process.
It is often assumed that if an animal lives longer it will age more slowly, but many creatures die from specific diseases rather than old age in general, scientists said.
For example, around 90 percent of mice die from tumors that form in their bodies as they grow old.
However, if the whole genome for factors that help mice to live long lives is examined, researchers would likely find many genes that suppress tumor development - and not necessarily ones that play a general role in aging, the experts said.
For the study, the team carried out a thorough health check in mice which looked at age-related changes to a wide range of bodily functions.
The check gave the team an “exact description” of the state of the animal at the time they were examined.
At different stages of life, the team compared how parameters changed at different stages of life and whether these changed more slowly when the creatures were given the treatments.
The team says the way the study was designed made it possible to work out whether the natural aging process can be slowed and the deterioration of important body functions can be slowed.
(Toasted Pictures via Shutterstock)
They said the results were “unambiguous” and showed that even when older mice appeared younger than they were, this had occurred for other reasons.
Dr. Dan Ehninger, from the German Centre for Neurodegenerative Diseases and an author of the study, said: “There is no internal clock of aging that you can regulate with a simple switch - at least not in the form of the treatments studied here.
“The fact that a treatment already has its effect in young mice – prior to the appearance of age-dependent change in health measures – proves that these are compensatory, general health-promoting effects, not a targeting of aging mechanisms."
The team now wants to look at the effects of other treatments experts believe can slow aging.
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